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Related post: Z01 AI 00286-09 LIP PERIOD COVERED October 1, 1989 to September 30 , 1990 TITLE OF Paxil Cr 37.5 Mg PROJECT (80 characters or less. Title must fit on one line between the borders.) Studies Of Genetic Control of Murine Leukemia Viruses and Virus-Induced Neoplasms PRINCIPAL INVESTIGATOR (List other professional personnel below the Principal Investigator.) (Name, title, laboratory, and institute affiliation) PI: J. W. Hartley Hd, Viral Oncology Section LIP, NIAID Others: M. Makino H.C. Morse III T.N. Fredrickson Visiting Fellow LIP, NIAID Laboratory Chief LIP, NIAID Research Microbiologist LIP, NIAID 6° 0P ^h N warT?zf? ny NIAID; D. Sachs, NCI, D. Murphy, NY State Dept. of Health; I. Egorov, Jackson Labs; C. David, Mayo Clinic; M. Zijlstra, Whitehead Inst. LAB/BRANCH Laboratory of Tmmnnopat-hol ogy SECTION Viral Oncology Section INSTITUTE AND LOCATION NIAID. NIH r Bethesda, MP 20892 TOTAL MAN-YEARS: 3.1 PROFESSIONAL: 1.6 1.5 CHECK APPROPRIATE BOX(ES) □ (a) Human subjects □ (a1) Minors □ (a2) Interviews □ (b) Human tissues S (c) Neither SUMMARY OF WORK (Use standard unreduced type. Do not exceed the space provided.) LP-BM5 MuLV induces in C57BL and other sensitive mouse strains a syndrome .of lymphoprol iteration and severe immunodeficiency, termed "MAIDS". Paxil Cr 37.5 " The causative virus is a replication-defective MuLV which usually requires "help" by competent B-tropic MuLVs, for which Fv-lb mouse strains, like C57BL, are permissive. Strain distribution patterns in inbred mice for sensitivity and resistance to MAIDS indicate major host influences associated with alleles at Fv-1, genes of the H-2 complex, and non-MHC related genes. Influences on disease reflect regulation of helper virus infection and spread, class I MHC control, and class II MHC Paxil Cr Coupon associated steps in pathogenesis. The most sensitive mice among Fv-lb strains are of the H-2b haplotype (H-2 haplotypes f,j,k,p,q,r and s are also associated with sensitivity) while Fv-lb H-2d strains are highly resistant. Mapping studies using intra-H-2 recombinants and transgenic mice indicate that H-2d-mediated resistance is clearly linked to the H-2D end of the region if the rest of the MHC is derived from the b haplotype. Expansion of these studies to explore the mechanism of D end resistance, to evaluate other regions of the MHC, and to clarify the pathogenesis of MAIDS reveal a very complex Paxil Or Paxil Cr interaction Paxil And Paxil Cr of H-2 region genes, both class I and class II in determining degree of sensitivity or resistance. For example, F-l hybrids of sensitive and resistant strains are sensitive, suggesting a Class II-like susceptibility; mice of s or q haplotype which are d Buy Paxil Cr at H-2D are not resistant although perhaps not fully sensitive; and mutations in major histocompatibility genes at H-2K can result in acceleration or delay in the disease process. Despite the B-tropism of the ecotropic helper virus in LP-BM5 certain Fv-ln strains have been found to be relatively sensitive to MAIDS, the mechanism apparently involving utilization of endogenous MuLV. PHS 6040 (Rev. 1/84) 9-9 DEPARTMENT OF HEALTH AND HUMAN SERVICES - PUBLIC HEALTH SERVICE NOTICE OF INTRAMURAL RESEARCH PROJECT PROJECT NUMBER Z01 AI 00465-05 LIP PERIOD COVERED October 1, 1989 to September ^0 , 1990 TITLE OF PROJECT (80 characters or toss. Title must fit on one line between the borders.) Retrovirus-induced Murine Immunodeficiency Syndrome PRINCIPAL INVESTIGATOR (List other professionej personnel below the Principal Investigator.) (Name, title, laboratory, and institute affiliation) PI: Others: H. C. Morse III J. W. Buy Paxil Cr Online Hartley M. Makino G. Shearer J. R. Wunderlich K. L. Holmes S. K. Chattopadhyay Expert Chief Senior Investigator Visiting Fellow Investigator Investigator Investigator LIP, NIAID LIP, NIAID LIP, NIAID LEI, NCI
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